Vestibular Testing (VNG, vHIT) Explained: Diagnosing Vertigo

Vestibular testing localises vertigo to inner ear, brain, or other systems. Learn VNG, vHIT and VEMP roles and interpretation.

By Elements84 Medical Editorial TeamFeb 15, 2026 8 min readReviewed by
Vestibular Testing (VNG, vHIT) Explained (vestibular testing vng) — ng vng vertigo explained illustration
Quick Answer

Vestibular testing localises vertigo to peripheral (inner ear) or central (brain) causes. Videonystagmography (VNG) records eye movements during positional manoeuvres, tracking, and caloric stimulation. Video head impulse test (vHIT) assesses semicircular canal function; VEMPs assess otolith function. Bedside HINTS exam distinguishes acute peripheral from central vertigo more reliably than early MRI.

Quick Reference

Vestibular testing is a suite of physiological tests to assess balance function. VNG records eye movements via infrared goggles during: oculomotor testing (saccades, smooth pursuit, optokinetic), positional testing (Dix-Hallpike identifies BPPV), spontaneous and gaze-evoked nystagmus, and bithermal caloric stimulation (unilateral caloric weakness suggests unilateral vestibular hypofunction, e.g., vestibular neuritis). Video head impulse test (vHIT) uses high-speed cameras to detect corrective saccades during rapid head thrust — normally the eyes stay locked on target; if vestibulo-ocular reflex fails, catch-up saccades appear. Cervical (cVEMP) and ocular (oVEMP) vestibular-evoked myogenic potentials assess saccule and utricle function respectively — abnormal in Ménière and superior semicircular canal dehiscence. In ACUTE vertigo, the bedside HINTS exam (Head Impulse, Nystagmus, Test of Skew) outperforms early MRI (98% sensitivity for stroke vs 88% for MRI-DWI at < 24 h). Central signs include: direction-changing nystagmus, vertical nystagmus, skew deviation, normal head impulse test. MRI is indicated if central signs, red flags, or suspected posterior circulation stroke.

Key Facts
VNG
Records eye movements, calorics
vHIT
Semicircular canal function
VEMPs
Otolith function
BPPV diagnosis
Dix-Hallpike + Epley
HINTS
Bedside test for acute vestibular stroke

Peripheral vs central acute vertigo signs

SignPeripheralCentral
Nystagmus directionUnidirectionalDirection-changing or vertical
Head impulse testAbnormalNormal
Skew deviationAbsentPresent
Hearing lossCommon (labyrinthitis)Rare
Neurological signsAbsentMay be present

When to order

Recurrent or persistent vertigo unexplained by BPPV.

Suspected Ménière disease.

Post-neuritis vestibular hypofunction quantification.

Superior semicircular canal dehiscence workup.

Balance rehabilitation planning.

Get seen promptly if
  • Acute vertigo + central signs — stroke pathway.
  • Sudden sensorineural hearing loss + vertigo — urgent ENT.
  • Vertigo + severe headache + neck pain — vertebral dissection workup.

Vertigo workup

  1. 1
    Positional + < 60 s + Dix-Hallpike positive?
    BPPV — Epley manoeuvre.
  2. 2
    Acute persistent + normal HINTS?
    Vestibular neuritis.
  3. 3
    Central signs on HINTS?
    MRI brain + neurology.
  4. 4
    Recurrent + hearing loss + tinnitus?
    Consider Ménière — VEMP + VNG.

Frequently asked questions

Key takeaways
  • VNG, vHIT and VEMPs localise vestibular dysfunction.
  • HINTS outperforms early MRI in acute vertigo.
  • BPPV is diagnosed by Dix-Hallpike and treated by Epley.
  • Central signs mandate MRI.
  • Vestibular rehab improves chronic symptoms.

References

2 sources
  1. AAO-HNS Clinical Practice Guideline: BPPV (2017 update)

    US ENT society.

    journals.sagepub.com

  2. Barany SocietyDiagnostic criteria for vestibular disorders

    International vestibular society.

    thejvr.com

VNGVertigoVestibularBPPV
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